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NCC Certified - Electronic Fetal Monitoring Sample Questions (Q63-Q68):

NEW QUESTION # 63
Uterine contraction intensity is manually measured by degree of uterine:

Answer: B

Explanation:
Comprehensive and Detailed Explanation From NCC-Aligned Equipment Concepts:
When using external tocodynamometry, uterine contraction intensity cannot be measured in mmHg. It is assessed manually, using palpation. NCC and AWHONN teach:
* Contraction intensity is estimated by palpating the fundus during a contraction.
* The degree of firmness versus indentation determines intensity:
* Mild # uterus easily indented
* Moderate # firm, difficult to indent
* Strong # rigid, cannot be indented
Why the incorrect answers are wrong:
* B. Muscle strength - Not measurable by external or manual exam.
* C. Pain - Not a reliable indicator; pain perception varies widely and does not correlate with uterine intensity.
Thus, the correct manual measurement is done through uterine indentation, making A correct.
References:NCC C-EFM Candidate Guide; AWHONN Principles & Practices; Menihan EFM; Miller's Pocket Guide; Simpson & Creehan.


NEW QUESTION # 64
A pattern of recurrent variable decelerations would move from Category II to Category III if what fetal heart rate change occurs?

Answer: B


NEW QUESTION # 65
A 20-year-old woman (G1P0) at 40-weeks gestation was admitted for cervical ripening with dinoprostone (Cervidil) four hours ago. She developed the pattern shown one hour ago. She has been changed to a lateral position and given a fluid bolus, and the pattern continues. An appropriate intervention would be to:

Answer: B

Explanation:
Comprehensive and Detailed Explanation From Exact Extract-Based NCC C-EFM References:
The tracing shows tachysystole (more than 5 contractions in 10 minutes) with minimal variability and recurrent decelerations consistent with uteroplacental insufficiency caused by excessive uterine activity.
Dinoprostone (Cervidil) is a uterotonic prostaglandin, and one of its known complications is uterine tachysystole with Category II or III fetal heart rate patterns.
NCC/AWHONN guidance for tachysystole caused by prostaglandins:
* FIRST intervention: Remove the dinoprostone insert.
* Reposition the patient (already done).
* IV fluid bolus (already done).
* Consider terbutaline only if tachysystole persists after removal of the agent.
Since maternal repositioning and IV fluids have already failed, the next step is to remove the cervical ripening agent.
Why other answers are incorrect:
* A. Continue to observe - Never acceptable with tachysystole + fetal intolerance.
* B. Terbutaline - May be used after prostaglandin removal, not before.
Thus, the correct answer is C. Remove the dinoprostone insert.
References:NCC C-EFM Candidate Guide; AWHONN Fetal Heart Monitoring Principles & Practices; Menihan; Miller's Pocket Guide; NICHD Definitions; Creasy & Resnik.


NEW QUESTION # 66
A fetal heart rate pattern shows no accelerations or decelerations. It would be interpreted as a Category II pattern if it occurred with:

Answer: C

Explanation:
Comprehensive and Detailed Explanation From NCC-Aligned Sources:
NICHD Category II includes:
* Minimal variability
* Marked variability
* Absent accelerations without recurrent decelerations
* Indeterminate baseline characteristics
A tracing with no accelerations and no decelerations becomes Category II if paired with marked variability, because marked variability indicates potential stress.
Why other answers are wrong:
* A. FHR 110 bpm # normal baseline if variability normal.
* B. Sinusoidal pattern # Category III, not Category II.
Correct answer: Marked variability.
References:NCC Candidate Guide; NICHD FHR Definitions; AWHONN FHMPP; Menihan.


NEW QUESTION # 67
Interventions undertaken to address fetal tachycardia are targeted at maximizing

Answer: C

Explanation:
Comprehensive and Detailed Explanation From Exact Extract NCC-Recommended Sources Fetal tachycardia is typically caused by maternal fever, dehydration, hypoxia, medications, infection, or fetal stress. AWHONN and Simpson & Creehan emphasize that management focuses on improving oxygen delivery across the placenta, which is governed by uteroplacental perfusion.
Menihan's EFM text states that "interventions for fetal tachycardia must address oxygen transfer by optimizing uteroplacental blood flow," including hydration, reducing uterine activity, maternal repositioning, and treating maternal fever.
Increasing maternal circulation alone is insufficient unless it improves placental blood flow. Enhancing fetal sympathetic tone is not a clinical goal and would worsen tachycardia.
Creasy & Resnik highlight that fetal heart rate abnormalities resolve when uteroplacental perfusion is restored, confirming this as the primary target of intervention.
References:
AWHONN - Fetal Heart Monitoring Principles & PracticesSimpson & Creehan - Perinatal NursingMenihan
- Electronic Fetal MonitoringCreasy & Resnik - Maternal-Fetal MedicineMiller's Pocket Guide


NEW QUESTION # 68
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